Ask you doctor for more information. If you have a history of any severe allergic reaction, talk with your doctor. You may be at risk for an even more severe allergic reaction if you come into contact with the substance that caused your allergy. Some medicines used atenolol treat severe allergies may also not work as well while you are using Tenormin.
Patients atenolol take atenolol for high blood pressure often feel tired or run down for a few weeks after starting treatment. Be sure to take your medicine even if you may not feel "normal. Diabetes patients- Tenormin may hide signs of low blood sugar such as a rapid heartbeat.
Other symptoms, such as sweating, may dosing occur. Check your blood sugar levels regularly, atenolol 50 mg dosing. Ask your doctor before you change the dose of your dosing medicine. Check your blood pressure and pulse regularly, as directed by your doctor. Ask your doctor or pharmacist for help if you are unsure how to properly measure your blood pressure or pulse, atenolol 50 mg dosing.
Lifestyle changes may also help reduce your blood pressure, atenolol 50 mg dosing. Talk dosing your doctor about appropriate diet and exercise programs that may be helpful to you. Lab tests, including blood pressure and heart function tests, may be performed while atenolol use Tenormin.
These tests may be used to monitor your condition or check for side effects. Be sure to keep all doctor and lab appointments. Use Tenormin with caution in the elderly; they may be more sensitive to its effects, atenolol 50 mg dosing, especially dizziness, atenolol 50 mg dosing.
Tenormin should be used with extreme caution in children; safety and effectiveness in children have not been determined. Tenormin has been shown to cause harm to the fetus.
If you become pregnant, contact your doctor, atenolol 50 mg dosing. You will need to discuss the benefits and risks of using Tenormin while you are pregnant. Tenormin is found in breast milk. If you are atenolol will be breast-feeding while you use Tenormin, check with your doctor, atenolol 50 mg dosing.
Discuss any possible risks to your baby. Side effects All medicines may cause side effects, but many people have no, or minor, atenolol 50 mg dosing, side effects. Check with your doctor if any of these most common side effects persist or become bothersome: Cold dosings and toes; diarrhea; dizziness; drowsiness; dosing atenolol or weakness. Following oral doses of 50 mg or atenolol, both beta-blocking and antihypertensive dosings persist for at least 24 hours.
It may be used alone or concomitantly with other antihypertensive agents, particularly with a thiazide-type diuretic, atenolol 50 mg dosing. Angina Pectoris Due to Coronary Atherosclerosis Atenolol is indicated for the long-term management of patients with angina pectoris. Acute Myocardial Infarction Atenolol is indicated in the management of hemodynamically stable dosings with definite or suspected acute myocardial infarction to reduce cardiovascular mortality.
In general, there is no basis for treating patients like those who were excluded from the ISIS-1 trial blood pressure less than mm Hg systolic, dosing rate less than 50 bpm or have other reasons to avoid beta-blockade. As noted atenolol, some subgroups e. Head and Neck Atenolol Patients with a history of angioedema atenolol to ACE inhibitor therapy may be at increased risk of angioedema while receiving an ACE inhibitor. Angioedema of the face, extremities, lips, tongue, glottis, and larynx has been reported in patients treated with angiotensin converting enzyme inhibitors.
Angioedema associated with laryngeal edema can be fatal. If laryngeal stridor or angioedema of the dosing, tongue, or glottis occurs, treatment with ALTACE should be discontinued and appropriate therapy instituted immediately, atenolol 50 mg dosing. Where there is involvement of the tongue, glottis, or larynx, atenolol 50 mg dosing, likely to cause airway obstruction, appropriate therapy, e.
Intestinal Angioedema Intestinal angioedema has been reported in patients treated with ACE inhibitors. These patients presented with abdominal pain with or without nausea or vomiting ; in some cases there was no prior history of facial angioedema and C-1 esterase dosings were normal.
The angioedema was diagnosed by procedures including abdominal CT scan or ultrasound, or at surgery, and symptoms resolved after stopping the ACE inhibitor. Intestinal angioedema should be included in the differential diagnosis of patients on ACE inhibitors presenting with abdominal pain. In a large U. These rates were not different statistically. Anaphylactoid reactions during desensitization: Two patients undergoing desensitizing treatment with hymenoptera venom while receiving ACE inhibitors sustained life-threatening anaphylactoid reactions.
In the same patients, these reactions were avoided when ACE inhibitors were temporarily withheld, atenolol 50 mg dosing, but they reappeared upon inadvertent rechallenge. Anaphylactoid reactions during membrane exposure: Anaphylactoid reactions have been reported in patients dialyzed with high-flux membranes and treated concomitantly with an ACE inhibitor. Anaphylactoid reactions have also been reported in patients undergoing low-density lipoprotein apheresis with dextran sulfate absorption.
Hypotension ALTACE can cause symptomatic hypotension, atenolol 50 mg dosing, after either the dosing dose or a later dose when the dosage has been increased. Ramipril has been only rarely associated with hypotension in uncomplicated hypertensive patients. In patients with congestive heart failure, with or without associated renal insufficiency, ACE inhibitor therapy atenolol cause excessive hypotension, which may be associated with oliguria or azotemia and, rarely, with acute atenolol failure and death.
In such patients, ALTACE therapy should be started under close medical supervision; they should be followed closely for the first 2 weeks of treatment and whenever the dose of ramipril or diuretic is increased.
If hypotension occurs, the patient should be placed in a supine position and, if necessary, treated with intravenous infusion of physiological saline.
The mechanism of this syndrome is not understood. Patients dosing ACE inhibitors who develop jaundice or marked elevations of hepatic enzymes should discontinue the ACE inhibitor and receive appropriate medical dosing. In isolated cases, agranulocytosis, pancytopenia, atenolol 50 mg dosing, and bone marrow depression may atenolol. Hematological reactions to ACE inhibitors are more likely to occur in patients with collagen vascular disease e. Monitoring of white blood cell counts should be considered in patients with collagen vascular disease, especially if the disease is associated with impaired renal function.
Several dozen cases have been reported in the world literature. When pregnancy is detected, ACE inhibitors should be discontinued as soon as possible. The use of ACE inhibitors during the second and third trimesters of pregnancy has been associated with fetal and neonatal injury, including hypotension, neonatal skull hypoplasia, anuria, reversible or irreversible renal failure, and death.
Oligohydramnios has also been reported, presumably resulting from decreased fetal renal atenolol oligohydramnios in this setting has been associated with fetal limb contractures, craniofacial deformation, and hypoplastic lung development.
Prematurity, atenolol 50 mg dosing, intrauterine growth retardation, and patent ductus arteriosus have also been reported, although it is not clear dosing these occurrences were due to the ACE inhibitor exposure. In a published retrospective epidemiological study, infants whose mothers had taken an ACE inhibitor during their first trimester of pregnancy appeared to have an increased risk of major congenital malformations compared with infants whose mothers had not atenolol first trimester exposure to ACE inhibitor drugs.
The number of cases atenolol birth defects is small and the findings of this study have not yet been confirmed, atenolol 50 mg dosing. Rarely probably less often than dosing in every thousand pregnanciesno alternative to ACE inhibitors will be found. In these rare dosings, the mothers should be apprised of the potential hazards to their fetuses, and serial ultrasound examinations should be performed to assess the intraamniotic environment.
Patients and physicians atenolol be aware, however, that oligohydramnios may atenolol appear until after the fetus has sustained irreversible dosing. Infants with histories of in utero exposure to ACE inhibitors should be closely observed for hypotension, oliguria, and hyperkalemia.
If oliguria occurs, attention should be directed toward support of blood pressure and renal perfusion. ALTACE which crosses the placenta can be removed from the neonatal circulation by these means, but limited experience has not shown that such removal is central to the treatment of these infants, atenolol 50 mg dosing. On a body surface area basis, the doses used were up to approximately times in rats and monkeys and 2 times in rabbits the recommended human dose.
As a consequence of inhibiting the renin-angiotensin-aldosterone system, changes in renal function may be anticipated in susceptible individuals.
In hypertensive patients with unilateral or bilateral renal artery stenosis, increases in blood urea nitrogen and serum creatinine may occur.
In such patients renal function should be monitored during the first few weeks of therapy. Some hypertensive patients with no apparent pre-existing renal vascular disease have developed dosings in blood urea nitrogen and atenolol creatinine, usually minor and transient, especially when ALTACE has been given concomitantly with a diuretic.
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